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A cerebral infarction is called a "stroke" in common parlance!

Clinical medications used to dissolve cerebral infarcts have been available for a long time!

Some stroke patients, if sent to the hospital in time, two injections of "alteplase" immediately turned into a miracle, paralyzed limbs can immediately move. It is as if a tripped switch has been reset and the wires have been reconnected.

Drugs commonly used clinically to dissolve cerebral infarction include urokinase, streptokinase, and alteplase.

Alteplase is a thrombolytic drug, but this drug is imported, very expensive, it seems that one is more than 4,000, usually use two, clinically used mainly for patients with cerebral infarction, stroke, but also can be used for cardiac infarction, pulmonary infarction dissolution, etc..

Previously in the neurology standardized training time to see a lot of stroke hemiplegia patients, if relatively early to the hospital (from the discovery of limb weakness to the hospital less than 4.5 hours), even if the patient has been completely paralyzed without consciousness, but the director of the Department of Neurology as long as the injection of two "alteplase" immediately turned into a miracle, the paralyzed limbs immediately can be moved. The paralyzed limbs could be moved immediately. It was as if a tripped switch had been reset and reconnected, and after a few days of treatment, the patient was as normal as if he had never had a stroke.

However, in real life, many patients with cerebral infarction occur suddenly in the middle of the night while they are sleeping, and the family members are only found in the morning, and it has long been more than 4.5 hours from the time the family members found out to the time they were sent to the hospital. So clinically there are still many patients with hemiplegia stroke.

Therefore, if you find that your family members have sudden weakness of limbs, hemiplegia, can't speak, crooked mouth, slurred speech, coma, etc., you should be sent to the hospital for further examination as fast as possible, and to a big hospital.

Below is a picture to recognize a stroke. If any of the following happens to someone in your family, please send them to the neurology department of a major hospital.

Since this is interprofessional science, I'm just speaking in general terms. Of course, there are now surgical removal of blood clots and surgical recanalization of blood vessels, but all of these need to be sent to a major hospital in a timely manner in order to do so.

Family members who find out that they have had a stroke should "get to the hospital as quickly as possible is the priority".

A comparison of three treatments for ischemic stroke, which is superior?

Associate Chief Physician, Neurosurgery Department, People's Hospital of Taojiang County, China

Cerebral infarction is caused by the narrowing of the arteries supplying blood to the brain, or the dislodgment of emboli from elsewhere blocking the cerebral blood vessels leading to cerebral ischemia. Most cerebral infarctions are caused by the occlusion of the arteries within the brain due to thrombosis and thromboembolism. InIn the early stage of cerebral infarction, the infarcted area is reversibly necrotic, and if the cerebral blood flow is restored in time and the metabolism of the brain tissue is improved it is possible to save the semi-dark band of tissue around the cerebral infarction and avoid the formation of permanent necrosis (cerebral infarction). Therefore, revascularization is the best treatment at present. Revascularization can be achieved by intravenous thrombolysis, arterial thrombolysis and arterial thrombolysis., (endovascular treatments include: arterial thrombolysis, mechanical thrombolysis and emergency angioplasty).

intravenous thrombolysis

There is now definitive evidence that intravenous thrombolysis of patients with acute cerebral infarction with rt-PA (recombinant tissue-type fibrinogen activator) applied within 4.5 hours of the onset of the attack (the time window) reduces the chances of severe disability and death and also greatly improves the quality of life of the patient. rt-PA dissolves blood clots, but it is necessary to require that the drug be injected into the patient's body through an The most significant risk of rt-PA treatment is the possibility of severe brain hemorrhage, which occurs in about 1 in 15 cases, and even sometimes leads to death, although the rate of death is much lower. If IV thrombolysis is given to eligible patients within 3 hours of the onset of the disease, the number of people who benefit from it is more than 10 times the number of people who have a severe hemorrhage; if it is given within 4.5 or 6 hours of the onset of the disease, the benefits still outweigh the disadvantages. For the total patient population, the potential benefits of this treatment far outweigh the risks. However, the individual patient needs to make his or her own decision about whether or not to undergo this treatment. Intravenous thrombolytic therapy requires that the patient arrive at the hospital in the shortest possible time and undergo a series of diagnostic procedures, investigations, and medications within 4.5 or 6 hours (preferably 3 hours) of the onset of symptoms. The earlier the patient arrives at the hospital, the more effective the treatment will be and the less chance of brain hemorrhage. It has the advantage of simplicity and time-saving, but the recanalization rate is low. Intravenous thrombolysis for acute ischemic stroke within 4.5 hours or 6h of onset is safe and effective, with patients treated with rt-PA within 3h of onset benefiting the most.

arterial thrombolysis

Arterial thrombolysis is a minimally invasive neuroradiological interventional endovascular procedure, which is a super-selective arterial thrombolysis based on intravenous thrombolysis with neuroradiological intervention. Theoretically, arterial thrombolysis has a higher rate of revascularization than intravenous thrombolysis, and also has the advantages of high local concentration and small dose of thrombolytic drugs. Arterial thrombolysis has been shown to be effective in patients with embolization of the internal carotid system, including the middle cerebral artery, within 6 hours of onset. For patients with basilar artery thrombosis, because the mortality rate after basilar artery thrombosis is very high, arterial thrombolysis may also be an effective salvage method, so patients with basilar artery occlusion may benefit from arterial thrombolysis within a certain period of time (currently believed to be up to 24 hours), and thus the time window and indications for arterial thrombolysis may be appropriately relaxed. However, arterial thrombolysis has specific risks and complications, whether patients can benefit from arterial thrombolysis depends on the development of the patient's specific condition. Arterial thrombolysis is performed by microcatheterization in the vicinity of the thrombus or through the thrombus to give thrombolytic drugs directly, improve the concentration of the local drugs, reduce the amount of drugs, and reduce the risk of intracranial and systemic hemorrhage; however, the method is time-consuming to a long period of time, and some of the embolus is difficult to dissolve the drugs.

mechanical takedown

Revascularization in the acute phase of cerebral infarction is the key to reducing lethality and disability. Traditional intravenous thrombolysis is effective in large vessel occlusion, but has a higher risk of bleeding, more contraindications, and a narrower time window, resulting in limited actual benefit to patients.

Stent mechanical thrombolysis is currently the most advanced technical means of treating acute cerebral infarction, and compared with thrombolytic therapy, it has the advantages of high safety, significant effect and long time window.It has been popularized in large medical centers both at home and abroad. Stent mechanical thrombus extraction is a guide wire with a diameter of about 0.3 mm is threaded from the patient's femoral artery into the body, and under fluoroscopic monitoring through the thrombus in the large blood vessels in the brain for localization, and then the microcatheter will be used to deliver the nickel-titanium alloy stent of the thrombus extraction stent to the site of the thrombus, and after releasing the stent, the nickel-titanium alloy stent will automatically expand and unfold to form a cylindrical mesh structure, which will be adhered with the wall of the vessel and embedded in the thrombus. The operator withdraws the thrombus extraction device, and the thrombus is pulled out of the body at the same time.

Mechanical thrombolysis has the advantages of high revascularization rate, low incidence of cerebral hemorrhage, recanalization time, and prolonged treatment time window. At present, many scholars believe that as long as MRI shows the existence of a large ischemic semi-dark band, the vessel should be actively opened, but the treatment time window needs to be further studied. Mechanical thrombolysis also has the disadvantages of delayed vessel recanalization due to surgical preparation and operation, and higher requirements for operators and equipment.

Several methods were mentioned, which one is the best? Let's start with an example, an embolus blocks a blood vessel, what to do, there are several ways, the first appeared in a very far away place with a corrosive (side effect of drugs, bleeding) drug to dissolve it, and later with people who thought that by putting this corrosive drug near or in the middle of the embolus, the blood vessel will certainly be better, and also reduce the effect of the drug on other blood vessels on the way through, which is the arterial thrombolysis, and then there were smarter people who thought wouldn't it be even better if they could just take the embolus out? This is mechanical thrombolysis, so theoretically speaking, it should be mechanical thrombolysis is the best effect, but this mechanical thrombolysis is the most technically demanding.

How is it? You think this article is helpful to you, please do not forget to click on the word "attention" and then "like" ah, but also can be forwarded to help other people in need!I am a professional neurosurgeon who specializes in interventional medicine, and will have more health knowledge about cerebrovascular disease to "dedicate" to you. Thank you.

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A little late to answer, but also better to answer and have an attitude.

Cerebral infarction means cerebral infarction, ischemic necrosis of the brain. Cerebral infarction can be caused by thrombosis of local blood vessels or by embolisms from distant places running into the cerebral blood vessels. Cerebral infarction itself can not be dissolved, because brain (nerve) cells can not be regenerated after necrosis, and there is no drug can be brought back to life, so once a cerebral infarction is formed, the brain cells are dead, and the function is gone.

Brain cells are very delicate, they rely on glucose to supply energy (that's why people will fall unconscious if their blood sugar is low!). The glucose stored in the brain is only enough to sustain for about 5 minutes, and to produce glucose, you need oxygen, which is supplied by the blood, so once the blood supply to the brain stops for more than 6 minutes, the brain cells will be necrotic because of the lack of energy, irreversibly necrotic, which is the reason why cardiopulmonary resuscitation in cardiac arrest has to be carried out in 4 minutes to have a high rate of resuscitation.

Back to the main topic, the brain infarction can not be dissolved, cerebral thrombosis can be dissolved (only thrombus Oh), but must be early, to catch up in the ischemic region of the brain tissue is not completely infarcted before the dissolution of thrombus, in order to maximize the salvation of ischemic dying brain tissue, the earlier the better (3 hours, 4.5 hours, more than 6 hours will not work).

There are thrombolytic drugs to dissolve blood clots, such as alteplase and urokinase, but they also need to be administered early in the course of the disease, and there are some requirements as to whether they can be administered. There are also techniques for removing blood clots from larger cerebral vessels and stenting of cerebral vessels, but these require certain medical conditions and must be performed at an early stage of the disease. Nowadays, some hospitals have set up stroke units, which can provide systematic treatment management and training for stroke patients from emergency to rehabilitation, which is conducive to the treatment of stroke patients.

Once cerebral infarction occurs, there will be loss of brain tissue, so prevention must be the mainstay to prevent cerebral infarction from occurring. It is important to prevent and treat related diseases that can trigger cerebral infarction, such as hypertension, diabetes, hyperlipidemia, hyperhomocysteinemia, atherosclerosis, etc., and to prevent and treat arrhythmias like atrial fibrillation and atrial flutter, which can cause blood clots in the heart (dislodgement).

(Image from the Internet)

Hello, I'm Dr. Knowles Blue.

Cerebral infarction, also known as ischemic stroke, gives the impression of hemiplegia, bedriddenness, and crooked mouth ...... That's right, these are the typical symptoms of cerebral infarction, and a lot of cerebral infarction patients have such sequelae. The formation of cerebral infarction is multifactorial, in which thrombus plays a very important role, so it is not difficult to understand, many people will have questions, is there any medicine can dissolve the thrombus? Today we will find out more about cerebral infarction and dissolving blood clots.

1. What causes brain infarction?

The essence of cerebral infarction is that the blood vessels supplying the brain tissue are blocked, causing ischemia, hypoxia, necrosis, and loss of function of the brain tissue, and such a change is a cerebral infarction. Why are the blood supply arteries blocked? There are several reasons:

First, atherosclerosisThe blood vessels of people are smooth inside when they are young, and as they get older, or with some bad lifestyle smoking, obesity, lack of exercise, high salt diet, high sugar diet, high fat cholesterol diet, etc., as well as some underlying diseases such as high blood pressure, diabetes mellitus, high blood cholesterol, and so on.

Second, pegs from other local sourcesFor example, our common carotid artery stenosis plaque, there is a possibility of plaque detachment, rushing to the cerebral blood vessels, causing blockage. Heart due to atrial fibrillation, heart failure is also prone to the formation of blood clots, blood clots with the blood flow may flow to the brain, causing blood vessel blockage, of course, may also block other parts of the body blood vessels, such as the upper limbs, abdominal cavity, lower limbs, etc., where the blockage where the ischemia.

2. Can the thrombus be dissolved?

The answer is yes, but there is a time limit. It is easy to understand that when a thrombus is first formed, it is relatively soft and fluffy, and the longer the time, the stronger it becomes and the less easy it is to dissolve, just like the cement for painting a wall, when it is first painted on, it can be washed off with water, but after a long time, a shovel can't even shovel it off. The best time to dissolve the thrombus is within 3 hours of the occurrence of cerebral infarction, if more than 6 hours, the effect of thrombolysis will be greatly reduced.

The most basic form of thrombolysis is by intravenous infusion of thrombolytic drugs such as urokinase and alteplase into the blood vessels, and as the blood circulates, a portion of it can reach the place of the blood clot. In medical units with conditions, thrombolysis can also be done by arterial thrombolysis, that is, a catheter is inserted from the artery in the thigh, and the arteries of the whole body are communicating with each other, and this tube can ultimately reach the place of cerebral artery thrombosis, and thrombolytic drugs can be injected into the place of thrombosis through this catheter, in this way the local concentration is higher, and the thrombolysis effect will be better.

3. Can all blood clots be dissolved?

As mentioned earlier, the effect of thrombolysis has a great deal to do with the timing of thrombolysis, and even if the same thrombolysis time is used for each person, the effect achieved may be different due to individual differences. In addition, sometimes it is not only the thrombus that clogs the blood vessel, but also the arterial plaque, and if the thrombus is dislodged from the heart, it is not known when the thrombus was formed, and there is also a relationship with the size of the thrombus.

4. Can all people be thrombolyzed?

This is not necessarily the case. First of all, some people come to the clinic when it has been a long time, thrombolysis is not very meaningful, thrombolysis itself is also risky, so the risk outweighs the benefit, can not be thrombolysis. In addition, some people have contraindications to thrombolysis, such as uncontrollable high blood pressure (greater than 180/110mmhg), a history of cerebral hemorrhage, recent major internal surgery, gastrointestinal bleeding and so on. Therefore, thrombolysis needs to be used only under the guidance of an experienced physician.

5. What are the risks of thrombolysis?

In fact, thrombolysis is a double-edged sword, on the one hand, want to dissolve the thrombus, on the other hand, is afraid of "accidentally injuring" themselves, causing bleeding, such as gastrointestinal bleeding, brain hemorrhage, etc., so bleeding is the first major risk. Another risk is ischemia reperfusion injury, aggravate brain damage, this term is more difficult to understand, let's say that the land in the field has been very dry, are cracked, the crops are withered, if you slowly pour a little bit of water crops may still live, if suddenly a lot of water into the crops will be "rotten roots", can not live.

In conclusion, cerebral infarction and thrombus are highly related, and thrombolysis is an important treatment for acute cerebral infarction, which can reduce death and disability and improve the quality of life. However, it is important to choose a good time and exclude those situations that are not suitable for thrombolysis so as not to increase the risk. Cerebral infarction is important to prevent, indeed after the unfortunate occurrence of cerebral infarction, the most critical thing is to seek medical treatment as soon as possible, the sooner the better.

1.If you are talking about the cause of cerebral infarction, there are medications that can dissolve it. Most cerebral infarctions are caused by a blood vessel that is blocked by a blood clot. Drugs that can dissolve blood clots can then reopen this blocked blood vessel and reperfuse the ischemic and hypoxic brain tissue.

Such thrombolytic drugs commonly used in the clinic are urokinase, streptokinase, Rt-peptidase, and so on.

But is it true that thrombolysis can be used whenever there is a cerebral infarction or that thrombolytic therapy can completely reopen the blood vessels? The answer is no, thrombolysis has a strict time limit, beyond the time of thrombolysis, the risk of cerebral hemorrhage is greatly increased. Some of them are other fallen embolus, the thrombolytic effect is not good, or the thrombus is too big and too long will also make the thrombolytic effect is not good.

Thrombolysis is generally categorized into intra-arterial thrombolysis and intra-venous thrombolysis, and interventional thrombolysis can also be used for large artery thrombosis if thrombolysis is not effective. The main risk of thrombolysis is reperfusion injury of the brain, as well as the risk of hemorrhage in various parts of the brain.

2.If you are talking about brain tissue that has been completely infarcted, there really is no effective drug to restore its fire, and the necrotic brain tissue ends up being dissolved and absorbed as well as gliosis to form soft foci of the brain.

3.If the brain tissue is temporarily ischemic and hypoxic but not necrotic, the medical term is called "ischemic semi-dark zone", through the reopening of the blocked blood vessels to improve cerebral perfusion, to relieve the necrotic brain tissue as well as the edema of the compression, and to remove the production of garbage metabolic components such as free radicals, the brain tissue of "ischemic semi-dark zone" can be restored. The brain tissue in the "ischemic semi-dark zone" can be restored.

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Cerebral infarction is a very serious type of disease, and many patients suffer from severe limb movement disorders due to very large cerebral infarction, which may even lead to death.

Today, Dr. Small Eyes explains the treatment options for cerebral infarction.

acute cerebral infarctionIn the case of the disease, thrombolytic therapy can be used to achieve the effect of opening the blood vessels.

As with myocardial transection, acute cerebral infarction, if your arterial plaque is formed acutely and is a white thrombus, fibrinoid thrombus, it can be treated with acute thrombolysis, which leads to reopening of the blood vessel and placing the onset of brain cell death.

However, if the thrombus is platelet-forming, thrombolytic therapy is not actually able to achieve a full therapeutic effect. Common thrombolytic drugs are able to dissolve the thrombus acutely to achieve the therapeutic effect.

In addition, with the gradual development of technologyStents for cerebral arteries are beginning to be gradually introduced in the clinic.

As with acute myocardial infarction.Patients with cerebral infarction can also be treated with acute dilatation of cerebral arteries and placement of stents to open up the blood flow in the cerebral vessels and to provide blood intoxication to the brain cells, avoiding severe cerebral infarction.

Clinical medication remains the top priority in the treatment of cerebral infarction.

According to large-scale clinical studies, it has been found that long-term adherence to regular medication does achieve a reduction in mortality and improvement in hospitalization rates. Therefore.Medication is the key to cerebral infarction treatment.

These oral medications includeAspirin enteric-coated tablets, clopidogrel medication, statins, all of these are key in the treatment of cerebral infarction. In addition, methylcobalamin tablets can also assist in the treatment to achieve the therapeutic effect.

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Thank you! In fact, in clinical practice, there are drugs that can dissolve clots, such as urokinase in the early years, and at the present time, the more common application is alteplase.

But you must also pay attention to a variety of things so that you can better solve the problem.

For example, the first is the problem of time, the general requirements for emergency thrombolysis is within 6 hours of the onset of thrombosis, that is to say, for fresh thrombus, to take the relevant drugs can achieve an effective thrombolysis of the therapeutic purpose, but for some of the onset of more than 6 hours, or even a few days a few months of this kind of stale thrombus, there is no special method can be effective treatment. So this point in time must pay attention to the suspicion of cerebral infarction immediately sent to the hospital for treatment, which can be more conducive to the stabilization of the condition in the later stage.

The second is whether there is any other change in the nature of bleeding inside the body, such as surgery within three months, etc., or the body is often bleeding gums bleeding urine bleeding and some other cases. Because this drug into the body, it is not like precision-guided missiles specifically targeting blood clots, it may travel throughout the body in all directions, and if there is a tendency to bleed patients, then it is likely to induce bleeding again, or even cause greater damage, so it is not worth the loss. Even in the clinic there are people who have kidney bleeding after taking thrombolysis.

The third is whether one's general vital signs are under active control, for example, whether the state of hypertension can be controlled relatively effectively, because if hypertension is not controlled at a relatively stable level and relevant medications are applied without authorization, hypertensive cerebral hemorrhage can easily be induced, which can lead to even worse clinical consequences.

The fourth one is to consider the problem of over-perfusion, which in layman's terms means that if you suddenly pour a big pot of water on a sapling that has already dried up, will it also be good for the sapling's growth? The brain is also, it has already formed a cerebral infarction, the end is already dry seedling, and now you open the blood vessels of the cerebral infarction, suddenly the blood rushes into the dry seedling, if the seedling can withstand, if it can or can, but if it can't withstand, then it may induce further necrosis, or even hemorrhage. Therefore, during thrombolysis, one must pay attention to the balance of one's blood pressure.

There are many cases of cerebral hemorrhage caused by thrombolysis, so it is important to pay attention to the occurrence of these things. In fact, this kind of problem is equivalent to brain hemorrhage, can apply hemostatic drugs to instantly block the bleeding blood vessels, he is currently speaking of the medical science is an unanswerable answer.

I hope everyone is in good health!

A cerebral infarction is said to be a cerebral infarction, which is an ischemic necrosis of the brain. It is partly caused by a localized blood clot, and also by a distant embolus that has traveled to a blood vessel in the brain.

In terms of cerebral infarction there is still no way to dissolve it, mainly because:

After the formation of cerebral infarction, brain cells die and cannot be regenerated: there is no way to resolve the cerebral infarction itself, because brain cells cannot be regenerated when they are necrotic, and there is no drug that can regenerate them. Up to now, there is no drug that can resolve cerebral infarction, and the brain cells lose their function when they are dead.

If the blood supply to the brain stops for more than six minutes, the brain cells die.

Glucose provides energy to the brain cells, the storage capacity of glucose on weekdays can only last for five minutes, the production of glucose requires oxygen, and oxygen needs to be supplied by the blood, so when the blood supply to the brain stops for more than six minutes, the brain cells due to the lack of energy will be necrotic and irreversibly necrotic, which is why cardiopulmonary resuscitation needs to be carried out within four minutes.

It's possible to dissolve a brain clot before it dies.

Although cerebral infarction can not be dissolved, but the cerebral thrombus still exists the possibility of dissolution, only must be early, need to be dissolved before all infarction, so that will maximize the possibility of saving the dying brain tissue. For the onset of not more than 4.5 hours, available thrombolytic drugs such as alteplase, such as rescue in time, and belongs to the temporary ischemia and oxygen, but not necrotic brain tissue, (medical become "ischemia semi-dark band"), by improving cerebral perfusion, eliminating necrotic brain tissue and oedema compression and garbage metabolic components, it is very likely to recover.

My mom is hemiplegic due to a cerebral infarction

The night of September 13th last year, I took the earliest high speed train back from Wuhan on the 14th, and have been taking care of her at home until now. When I saw her on the 14th, she was sleeping in the hospital bed and couldn't speak clearly, but her consciousness was clear, and the moment we looked at each other, we both cried and couldn't help ourselves! It was really heartbreaking to see a person who was usually so lively and active unable to get up when she was sick! At that time in the hospital room, my father and I were by her side, and then the family was too busy, my father went back first, I was alone in the hospital to take care of.

My mom suffered from severe cerebral infarction and collapsed three times before she was found and sent to the hospital. I heard that even the doctor thought she was dying on the day she was admitted to the hospital. After she was hospitalized, the attending doctor recommended us to use "Butylphthalide Injection" according to her condition. I remember it was two vials per day, one in the morning and one in the afternoon, and we had been injecting it for almost 10 days before we could stop the medicine. During this period, my mom recovered quite well, she could speak clearly, and her hemiplegic left foot had some contraction reaction. The doctor said that "butylphthalide" was the most effective medicine for treating cerebral infarction hemolysis in the hospital, and suggested us to take "butylphthalide capsule" after we were discharged from the hospital.

I have to say that cerebral infarction is really a very abrasive disease! We have been taking traditional Chinese medicine, western medicine, and physical therapy, and my mom can now walk on her own with crutches. Although she can't take care of herself completely, she's recovering quite well, after all, when she first went to the hospital, she was dying! Now that the weather is getting warmer every day, I hope that my mom will get better sooner, so that she will suffer less!

How so? Maybe in some ways, there are actually medications to defuse brain attacks!

First of all, brain infarction can be prevented with medication and lifestyle changes. As I said in my previous Q&A, advanced age, family history of smoking, alcohol consumption, overweight, hypertension, diabetes, high blood cholesterol, etc. are all risk factors for cerebral infarction, except for age and family history which we can't control, the others are all things we can change and control, and some medications, such as aspirin and statin, have the effect of preventing cerebral infarction, which, from the point of view of reduced probability of morbidity, cerebral infarction should be considered to have been "defused".

Secondly, in the acute phase of cerebral infarction, within 4.5 hours of the onset of the disease (in some cases, it can be up to 6 hours or longer), thrombolytic drugs such as alteplase, also known as recombinant tissue-type activator of plasminogen activator (rtPA) intravenous or arterial thrombolysis can be used, and if the rescue is timely, some of the brain cells that are in the "half ischemia" can be rescued, and this can be considered to have solved part of the cerebral infarction, right? This can also be regarded as a partial solution to cerebral infarction!

Of course, if a brain infarction has already formed, after all, some of the brain cells are not salvageable, and will form glial cell scars or soft foci, for this time of the brain infarction, there is indeed no medication can be "dissolved". Perhaps, with the development of medical technology in the future there will be a way to remodel brain function, but not yet.

So, it's best not to wait until a cerebral infarction occurs to defuse it, it's best to prevent it well before it occurs, or to emphasize, cerebral infarction"Prevention is better than cure!”

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